By Devin Binder, D. Sonne, Nancy Fischbein
This fantastically illustrated publication combines an in depth exposition of the anatomy and serve as of the cranial nerves with sensible assurance of medical options for the evaluate and differential prognosis of cranial nerve disorder. An introductory bankruptcy offers a short evaluate of cranial nerve anatomy and serve as, cranium base anatomy, type of pathologies, and imaging techniques. all of the twelve chapters that keep on with is dedicated to in-depth assurance of a unique cranial nerve. those chapters open with special dialogue of some of the capabilities of every nerve and basic anatomy. The authors then describe universal lesions and current a sequence of circumstances which are complemented through CT photos and MRIs to demonstrate affliction entities that bring about cranial nerve dysfunction.Highlights:-Concise descriptions in a bulleted define layout let fast examining and review-Tables synthesize key details regarding anatomy, functionality, pathology, and imaging-More than three hundred fine quality illustrations and cutting-edge CT and MR photographs reveal very important anatomic innovations and pathologic findings-Pearls emphasize medical details and key imaging findings for prognosis and treatment-Appendices comprise certain details on brainstem anatomy, student and eye circulation keep watch over, parasympathetic ganglia, and cranial nerve reflexesThis e-book is an crucial reference for working towards physicians and trainees in neurosurgery, neurology, neuroradiology, radiology, and otolaryngology-head and neck surgical procedure. it is going to additionally function a necessary source for college kids looking to achieve an outstanding knowing of the anatomy, functionality, and pathology of the cranial nerves.
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Extra resources for Cranial Nerves: Anatomy, Pathology, Imaging
Raldy mebstasize. s~ral different pathologic types (~e basic types: menlngotbellal, fibroblastic, and transitional) have been described. Grading: Grade I, benign; Grade D, atypical; Grade Dl, anaplastic/malignant. • ltartment. JeiY with or without preoperative embolizatiOII. Radiation therapy may be indicated lbr residual and/or =rrent disease. Prognosis u g~:nerally good. foJo&y. en percent of primary Intracranial tumors; peak at age 40 tD 60 years, and females are more COI1IIDOJily affected. Incidence increased by prior radiation and the p~sente of genetic syndromes sudl as neurofibromatosis type 2 (NF-2).
Neuroretinitis. • Popi/Utis is optic disc edema associated with inflammatory or demyelinating diseases. • Popil/edemais optic discedemaassodated with increased intracranial pressure. causes enlarged visual blind spot and constricted visual field without visual acuity change. Develops as increased cerebrospinal Duid (CSF) pressure in the optic sheath compresses nerve fibers, resulting in axonal swelling and leaking. Unilateral papilledema can occur with optic nerve tumors. Visual acuity and pupillary light reflexes are usually normal (especially early).
It is the first symptom of MS in 15% of cases. Vision returns to normal within a few weeks in two thirds of cases. Steroids speed the recovery (intravenous methylprednisolone followed by oral prednisone). Dyschromatopsia is a common persistent problem. In children, it is more frequently bilateral and of a viral or post-viral etiology. Differential diagnosis indudes MS, acute disseminated encephalomyelitis (ADEM), syphilis, Lyme disease, vasculitis, neurosarcoidosis, and prior radiation. • Ischemic optic neuropathy.