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By Herbert A. Blough, John M. Tiffany (auth.), W. Arber, W. Henle, P. H. Hofschneider, J. H. Humphrey, N. K. Jerne, P. Koldovský, H. Koprowski, O. Maaløe, R. Rott, H. G. Schweiger, M. Sela, L. Syruček, P. K. Vogt (eds.)

The methods concerned about herpesvirus replication, latency, and oncogenic transformation, have, ordinarily, been really poorly outlined. a main cause of this can be the scale and complexity of the herpesvirus genome. certainly, a greater knowing of the features of the viral genome in contaminated and remodeled cells should be accomplished via reviews with temperature-sensitive (ts) mutants of herpesviruses on account that, theoretically, any crucial gene functionality will be plagued by mutants of this kind. A. The Herpesviruses A attention of the genetic research of individuals of the herpesvirus team necessitates an outline, albeit short, of the houses of the crowd and, most significantly, in their genetic fabric. The herpesviruses contain a bunch of particularly huge (100-150 nm), enveloped viruses. The envelope surrounds an icosahedral capsid enclosing a center which includes double­ stranded DNA (ROIZMAN, 1969). the crowd is therefore outlined at the foundation of a typical virion morphology. as well as a typical constitution, contributors of the crowd proportion a few organic homes reminiscent of an identical replicative cycle, the power to reason latent and protracted infections, and the power to urge antigenic adjustments of contaminated cellphone membranes. a number of herpes­ viruses were linked lately with malignancies in guy and animals (KLEIN, 1972). Herpesviruses are ubiquitous and feature been defined in over 30 assorted species (HUNT and MELENDEZ, 1969; WILDY, 1971; FARLEY et aI. , 1972; KAZAMA and SCHORNSTEIN, 1972; NAHMIAS et aI. , 1972; ROlZMAN et aI. , 1973). Their frequent prevalence in nature indicates a typical ancestor.

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By Herbert A. Blough, John M. Tiffany (auth.), W. Arber, W. Henle, P. H. Hofschneider, J. H. Humphrey, N. K. Jerne, P. Koldovský, H. Koprowski, O. Maaløe, R. Rott, H. G. Schweiger, M. Sela, L. Syruček, P. K. Vogt (eds.)

The methods concerned about herpesvirus replication, latency, and oncogenic transformation, have, ordinarily, been really poorly outlined. a main cause of this can be the scale and complexity of the herpesvirus genome. certainly, a greater knowing of the features of the viral genome in contaminated and remodeled cells should be accomplished via reviews with temperature-sensitive (ts) mutants of herpesviruses on account that, theoretically, any crucial gene functionality will be plagued by mutants of this kind. A. The Herpesviruses A attention of the genetic research of individuals of the herpesvirus team necessitates an outline, albeit short, of the houses of the crowd and, most significantly, in their genetic fabric. The herpesviruses contain a bunch of particularly huge (100-150 nm), enveloped viruses. The envelope surrounds an icosahedral capsid enclosing a center which includes double­ stranded DNA (ROIZMAN, 1969). the crowd is therefore outlined at the foundation of a typical virion morphology. as well as a typical constitution, contributors of the crowd proportion a few organic homes reminiscent of an identical replicative cycle, the power to reason latent and protracted infections, and the power to urge antigenic adjustments of contaminated cellphone membranes. a number of herpes­ viruses were linked lately with malignancies in guy and animals (KLEIN, 1972). Herpesviruses are ubiquitous and feature been defined in over 30 assorted species (HUNT and MELENDEZ, 1969; WILDY, 1971; FARLEY et aI. , 1972; KAZAMA and SCHORNSTEIN, 1972; NAHMIAS et aI. , 1972; ROlZMAN et aI. , 1973). Their frequent prevalence in nature indicates a typical ancestor.

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LINDAHL. : Linear association between cell D~A and EpsteinBarr virus DNA in a human lymphoblastoid ce ll line . Proc. nat . Acad. Sci. ) 70, 2888-2895 (1973) ANDERSON , W. , KILHOUR1'E, E. : Induced reactivation of Herpes simplex virus in healed rabbit corneal lesions. Proc. Soc. expo BioI. ) 107, 628-631 (1961) ANOREWES, C. , CAR~ICHAEL, E. A . : A note on the presence of antibodies to herpes virus in post-encephalitic and other human sera . , GEERI:-'-G, G. , OLD , L. J . : In: Recent advances in human tumor virology and immunology.

J. Med. 246, 172-176 (1952) CLEOI3URY, J . , SKIK:-1ER, G . , THOULESS, M. : Association between psychopathic disorder and serum antibody to Herpes simplex virus (Type 1). Brit. med. J. 1, 438- 439 COOK, M. , BASTONE , V. , STEVENS, J. : Evidence that neurons harbor latent Herpes simplcx virus. Inf. and Imm. 9,946-951 (1974) COOK, 1\1. , STEVE:-1S, J. ' Pathogenesis of herpetic neuritis and ganglionitis in mice: evidence for intra-axonal transport of infection. Inf. and Imm. 7, 272- 288 (1973) CORIELL, L.

M. : Further observations on the structure of the lipid-containing bacteriophage PM2. ) 248, 681-682 (1974) SCHEID, A. , CHOPPIN, P . : Identification of biological activities of paramyxovirus glycoproteins. Activation of cell fusion , hemolysis, and infectivity by proteolytic cleavage of a n inactive precursor protein of Sendai virus . Virology 57, 475-490 (1974) Viral Envelopes: Structure and Assembly 29 SCHLESINGER , M. , BURGE, B. : Identification of a second glycoprotein in Sindbis virus. Virology 47, 539-541 (1970) SCHULZE, I.

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