By Susan Okie
Examines a few of the components contributing to early life weight problems, comparable to terrible vitamin, loss of workout, suburban sprawl, and television, and offers suggestion on how mom and dad could make optimistic alterations.
Read or Download Fed Up!: Winning the War Against Childhood Obesity PDF
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Extra resources for Fed Up!: Winning the War Against Childhood Obesity
When insulin resistance occurs, specialized cells in the pancreas called beta cells must churn out extra-high levels of the hormone in response to the rise in blood glucose that takes place when a person eats. Eventually the beta cells can’t make enough insulin to lower the blood glucose level to within the normal range. Chronically high glucose levels ultimately destroy beta cells, the only cells that can manufacture insulin. Over a period of years, type 2 diabetes causes just as much mayhem to tissues as type 1 diabetes.
The rest may be due to gene variants more common in one ethnic population than another, so that genes predisposing someone of Mexican descent to obesity might differ from those determining risk for Polynesians. “This is what makes the genetics so complicated,” Leibel says. “We don’t know which ones are the major players and we don’t know which ones are the minor players. ” Although genes determine individual vulnerability to weight gain, environmental factors help dictate the outcome—the weight that a person reaches during childhood or adulthood.
Obese people today are living in a time that is both promising and painfully difficult. Although doctors have begun to recognize that body size is to a great degree determined by a person’s genetic and biological vulnerability to weight gain and thus largely beyond an individual’s conscious control, the science of obesity is still young. Despite the rapid pace of new discoveries in the field—and the wealth of scientific talent and pharmaceutical and government funding now focused on it—specific, highly effective treatments for obesity have yet to be discovered.